T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t (1991)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; CITOLOGIA; BIOLOGIA CELULAR; ONCOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Basingstoke
- Date published: 1991
- Source:
- Título do periódico: Oncogene
- Volume/Número/Paginação/Ano: v.6 , p.1049-56, 1991
-
ABNT
RAMEH, L E e ARMELIN, M C S. T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t. Oncogene, v. 6 , p. 1049-56, 1991Tradução . . Acesso em: 10 jun. 2024. -
APA
Rameh, L. E., & Armelin, M. C. S. (1991). T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t. Oncogene, 6 , 1049-56. -
NLM
Rameh LE, Armelin MCS. T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t. Oncogene. 1991 ;6 1049-56.[citado 2024 jun. 10 ] -
Vancouver
Rameh LE, Armelin MCS. T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t. Oncogene. 1991 ;6 1049-56.[citado 2024 jun. 10 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
- Generation of polyclonal antiserum to the retinoblastoma protein (prb) to study the role played by tumor suppressor genes in c6 / st1 cells' transformed tumor to normal phenotypic reversion
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